gland health in several studies. Whole blood Se concentra- tion and glutathione peroxidase activity were negatively related to prevalence of intramammary infection in a study involving 32 dairy herds in Pennsylvania.High serum Se concentrations were associated with reduced rates of mastitis and lower bulk-tank somatic cell counts in Ohio dairy herds. evaluated the effects of supplemental Se and/or vitamin E on mastitis in cows fed diets low in both Se and vitamin E. Administration (IM) of 0.1 mg Se/kg BW at 21 days before calving did not affect the inci- dence of clinical mastitis but reduced the duration of clin- ical symptoms in cows with clinical mastitis by 46%. Selenium administration in combination with oral supple- mentation of vitamin E (740 IU/day) was most effective, reducing incidence of mastitis and duration of clinical symptoms by 37% and 62%, respectively .In this study, mastitis was caused by environmental pathogens (Coliform and Streptococcus and not
Staphylococcus aureus. Dietary Se supplementation to diets deficient in Se unknown vitamin E status) reduced the rate of new intramammary gland infections in dairy cows .Experimental mastitis, induced by intramammary challenge with Escherichia coli was more severe and of longer duration in cows receiving 0.04 mg Se/kg diet compared with those receiving 0.14 mg Se/kg diet. Following intramammary challenge with S. aureus, Selenium-deficient cows had greater peak bacteria concentrations in milk than Se-supplemented cows However, the severity and duration of infec- tion was not affected by dietary Se in this study. Numerous studies have indi- cated that pre-partum Se supplementation can reduce the incidence of retained placenta in dairy cows fed diets low in Se. Failure of supplemental Se in some studies to affect the incidence of retained placenta can be explained by the control diets being adequate in Se. Most studies have involved IM administration of Se in combination with vitaminE. However found that oral Se supplementation was as effective as IM administration of Se and vita-min E in preventing retained placenta in cows fed Se deficient diets. Cows with retained fetal membranes had lower glutathione peroxidase activity in maternal and
placental tissues than cows without retained
gland health in several studies. Whole blood Se concentra- tion and glutathione peroxidase activity were negatively related to prevalence of intramammary infection in a study involving 32 dairy herds in Pennsylvania.High serum Se concentrations were associated with reduced rates of mastitis and lower bulk-tank somatic cell counts in Ohio dairy herds. evaluated the effects of supplemental Se and/or vitamin E on mastitis in cows fed diets low in both Se and vitamin E. Administration (IM) of 0.1 mg Se/kg BW at 21 days before calving did not affect the inci- dence of clinical mastitis but reduced the duration of clin- ical symptoms in cows with clinical mastitis by 46%. Selenium administration in combination with oral supple- mentation of vitamin E (740 IU/day) was most effective, reducing incidence of mastitis and duration of clinical symptoms by 37% and 62%, respectively .In this study, mastitis was caused by environmental pathogens (Coliform and Streptococcus and not Staphylococcus aureus. Dietary Se supplementation to diets deficient in Se unknown vitamin E status) reduced the rate of new intramammary gland infections in dairy cows .Experimental mastitis, induced by intramammary challenge with Escherichia coli was more severe and of longer duration in cows receiving 0.04 mg Se/kg diet compared with those receiving 0.14 mg Se/kg diet. Following intramammary challenge with S. aureus, Selenium-deficient cows had greater peak bacteria concentrations in milk than Se-supplemented cows However, the severity and duration of infec- tion was not affected by dietary Se in this study. Numerous studies have indi- cated that pre-partum Se supplementation can reduce the incidence of retained placenta in dairy cows fed diets low in Se. Failure of supplemental Se in some studies to affect the incidence of retained placenta can be explained by the control diets being adequate in Se. Most studies have involved IM administration of Se in combination with vitaminE. However found that oral Se supplementation was as effective as IM administration of Se and vita-min E in preventing retained placenta in cows fed Se deficient diets. Cows with retained fetal membranes had lower glutathione peroxidase activity in maternal and placental tissues than cows without retained
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